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Adaptions of endurance training

Module

Physiological Response to Exercise (SPRT586)

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Academic year: 2019/2020
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Leeds Beckett University

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Adaptions of endurance training Adapations aren’t as great in younger individuals - Muries et al – young 18% (0/min )increase and old 31% increase (0/min) Qmax – arterial o2 content per 100ml of blood – 56% of young individuals increase in vo2 ma was explained by qmax – elderly was 69% a-vo2 – venous o2 conent per 100ml of blood – 34% exline by increased maximal a-vo2 – elderly was 31% left and right ventricular masses were 36 and 37% higher in trained athletes endurance atheltes have greater EDV of hypertrophied ventricles and not to higher ejection factor why do we see an increase in a-vo2 differeence – increases capilliary density summery of papers – how endurance training increases vo2max by  Increasing cardiac output via increased ventricle size, end diastolic volume = increased maximal SV  Increased plama volme and Hb  Increased o2 extraction via increased capillaries which increases transit time of blood

Activity of enzymes within the Krebs cycle increase with endurance training Increased oxidative capacity increased the activity of citrate synthase and succinate dehydrogenase Higher PFK enzyme activity in untrained atheltes Summery – endurance exercise training achieves physiological adaptions that  Increase maximal o2 consumption  Increase oxidative capacity  Increase the capacity to utilise fat  Decrease reliance on energy system and fue what results in rapid fatigue

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Adaptions of endurance training

Module: Physiological Response to Exercise (SPRT586)

9 Documents
Students shared 9 documents in this course
Was this document helpful?
Adaptions of endurance training
Adapations aren’t as great in younger individuals - Muries et al – young 18% (0.16L/min )increase
and old 31% increase (0.13L/min)
Qmax – arterial o2 content per 100ml of blood – 56% of young individuals increase in vo2 ma was
explained by qmax – elderly was 69%
a-vo2 – venous o2 conent per 100ml of blood – 34% exline by increased maximal a-vo2 – elderly was
31%
left and right ventricular masses were 36 and 37% higher in trained athletes
endurance atheltes have greater EDV of hypertrophied ventricles and not to higher ejection factor
why do we see an increase in a-vo2 differeence – increases capilliary density
summery of papers – how endurance training increases vo2max by
Increasing cardiac output via increased ventricle size, end diastolic volume = increased
maximal SV
Increased plama volme and Hb
Increased o2 extraction via increased capillaries which increases transit time of blood
Activity of enzymes within the Krebs cycle increase with endurance training
Increased oxidative capacity increased the activity of citrate synthase and succinate dehydrogenase
Higher PFK enzyme activity in untrained atheltes
Summery – endurance exercise training achieves physiological adaptions that
Increase maximal o2 consumption
Increase oxidative capacity
Increase the capacity to utilise fat
Decrease reliance on energy system and fue what results in rapid fatigue