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General Overview of Anxiety
Course: Health Care Concepts II (NSG 211)
45 Documents
Students shared 45 documents in this course
University: Germanna Community College
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Anxiety
exemplars: gad, panic disorder, phobias, ocd, ptsd
Overview: Anxiety is a natural survival response to stress, helping individuals avoid potential threats by
triggering feelings of fear, nervousness, or worry. It is often based on an anticipated future threat or
stressful event. Initially, anxiety enhances performance by increasing blood flow to the heart, muscles,
and brain, motivating the individual to take action. However, if anxiety becomes excessive or persistent,
it can escalate to panic, impairing daily functioning. Seyle’s General Adaptation Syndrome (GAS) explains
this physiological response to stress, showing how anxiety can shift from an adaptive mechanism to a
maladaptive one, resulting in both psychological and physical distress. When anxiety reaches this level, it
can be classified as an anxiety disorder, characterized by its disabling effects on normal life.
Anxiety and stress are often confused, but they are distinct responses. Stress typically occurs as a
proportional reaction to a specific situation, while anxiety is a reaction to stress and becomes a disorder
when it is inappropriate for the situation, happens too frequently, interferes with functioning, or lasts too
long.
Simplified Pathophysiology of Anxiety: Anxiety shares some symptoms with stress and depression, like
trouble sleeping, difficulty concentrating, and fatigue. The pathophysiology of anxiety starts with the
brain's fear center, the amygdala, and the hypothalamus, which trigger the body's survival responses
(fight, flight, freeze, fawn) by releasing stress hormones. This is controlled by the amygdala-centered
circuit, which drives fear and panic. Another brain loop, the cortico-striato-thalamo-cortical circuit
(CSTC), is linked to worry, obsessive thoughts, and anxiety. This loop involves neurotransmitters like
serotonin, GABA, dopamine, and norepinephrine, which play key roles in anxiety and are targets for
medications used in treatment.
Seyle’s General Adaptation Syndrome (GAS) outlines the body's predictable response to stress
through three stages:
1. Alarm Phase: The body enters a heightened state of alert when exposed to stress, initiating the
fight-flight-freeze-fawn response. Heart rate increases, cortisol is released, and the brain
becomes more focused on managing the stressor.
2. Resistance Stage: The body attempts to stabilize and recover from the alarm phase. While still
in a heightened state of alert, the autonomic nervous system gradually downregulates. If the
stressor resolves, the body returns to normal functioning; if it persists, the body remains
stressed.
3. Exhaustion Phase: If the stress continues without resolution, the body’s energy reserves are
depleted, leading to an inability to self-regulate effectively, resulting in physical and mental
exhaustion.
Hildegard Peplau’s Theory of Interpersonal Relations is foundational for the nurse-client therapeutic
relationship and outlines the nurse’s role in managing the client's anxiety. The theory describes four
phases of the relationship: orientation, identification, exploitation, and resolution, which guide
interactions.
Peplau also highlights different levels of anxiety: