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Med surg 2 blueprint exam 2

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Medical-Surgical Nursing II (NSG 223)

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NSG223 Medical-Surgical II

EXAM 2 STUDY GUIDE

Topic Location Student Notes

Pulmonary embolism

  • pathophysiology NSG 223.04.

▪ PE: refers to obstruction of the pulmonary artery or one of its branches by a thrombi that originates somewhere in the right side of the heart ▪ Causes: ▪ Trauma ▪ Surgery (orthopedic, abdominal, pelvic,..) ▪ Pregnancy ▪ Heart Failure ▪ Hypercoagulable states Prolonged immobility ▪ Most commonly PE is due to blood clot... can also be due to air, fat, amniotic fluid, sepsis (bacterial vegetation) ▪ Complete or partial occlusion ▪ Little or no blood-flow to distal area ▪ Ventilation/Perfusion impairment ▪ Impaired or no gas exchange ▪ Hemodynamic changes – edema, regional vasoconstriction – increased pressures – can lead to right sided heart failure

Pulmonary embolism - Clinical manifestations

NSG223.04.01 ▪ Depends on the size of the thrombus... ▪ Dyspnea ▪ Tachycardia ▪ Bloody sputum ▪ Cough ▪ Chest pain (pleuritic) ▪ Anxiety ▪ Fever ▪ Diaphoresis ▪ Syncope

Pulmonary embolism - Treatments

NSG223.04.01 ▪ Emergency: ▪ Nasal O2 immediately to relieve hypoxemia, respiratory distress, central cyanosis...

may need to intubate ▪ IV lines for fluids and meds-anticuagulants, vasopressors etc.. ▪ Hypotension – if not improved by IVF – give vasopressors (Dopamine, Dobutamine, Norepinephrine) ▪ Hemodynamic eval, Hypoxemia eval (ABG) and MDCTA ▪ EKG ▪ Labs – electrolytes, CBC, coagulation ▪ Pain and discomfort relief – small doses MS, sedatives if intubated ▪ General management: ▪ Oxygen therapy to relieve hypoxemia ▪ Anti-embolism stockings or SCDs to reduce venous stasis ▪ Elevate legs for venous return ▪ Fluid challenge for hypotension ▪ Anticoagulation therapy: ▪ Suspected PE – tx up to 10 days ▪ After embolic event - long-term tx – 10 days to 3 months ▪ With PE, but hemodynamically stable – LMW Heparin (Lovanox), unfractioned Heparin (sq), or new oral agent such as Dabigatran (Pradaxa), Rivaroxaban (Xarelto) ▪ If pt does not qualify for new oral agents – Coumadin – needs regular blood draws for INR – bleeding precautions – antidote is Vit K ▪ Thrombolytic therapy: ▪ Used in pt with acute PE, hemodynamically unstable, do not have any risk factors for bleeding ▪ Clot busters! – Activase, Streptase ▪ Pt must meet criteria! ▪ Labs done prior to starting tx – PTT, INR, Hematocrit, Platelets ▪ Anticoagulant: Heparin IV if pat doesn’t meet criteria for clot busters

Pulmonary embolism - Prevention

NSG223.04.01 ▪ ID the pt at risk ▪ Prevent thrombus formation ▪ Encourage ambulation ▪ Active and passive leg exercise ▪ Pumping exercise in bed or standing next to bed ▪ Do not sit or lie for prolonged periods ▪ Do not cross legs ▪ Do not wear constrictive clothing

ARDS

  • Mechanical Ventilation

NSG223.04.02 ▪ Management almost always includes INTUBATION – mechanical ventilation ▪ PEEP – critical – improves oxygenation by keeping alveoli from collapsing ▪ Goal PaO2 is > 60mmHg, and V/Q balance

ARDS - Nursing Care of Patients on Ventilators

NSG223.04.02 ▪ Watch for and treat hypovolemia ▪ Circulatory support – IVF, inotropic and vasopressor agents ▪ Sedation and paralysis ▪ Prone ▪ Nutrition

Mechanical Ventilators - Medications

NSG223.04.02 ▪ Sedatives – Lorazepam (Ativan), Midazolam (Versed), Propofol (Diprivan) ▪ Neuromuscular blocking agents – Pancuronium (Pavulon), Vecuronium (Norcuron) – often given prior to intubation to relax jaw and vocal cords ▪ Nursing care important – to check effectiveness of these agents – use “train of 4” – 4 consecutive stimuli delivered along nerve, check reaction

Mechanical Ventilators NSG223.04.02 See above***

Medications Pulmonary edema/failure - Nursing interventions

NSG223.04.03 ▪ MANAGEMENT: ID and correct underlying cause – O2, intubate, ventilate

Pulmonary edema/failure - Complications

NSG223.04.03 Respiratory Failure Complications ▪ Respiratory arrest

Pulmonary edema/failure - Treatments

NSG223.04.03 Pulmonary Edema ▪ Treatment: Depending on cause..... (Furesomide)-IV or PO depending on the severity Respiratory Failure Tx ▪ MANAGEMENT: ID and correct underlying cause – O2, intubate, ventilate

Management of Acid-base balance - ABG interpretation

NSG223.05.

Management of Acid-base balance - Treatments for each ABG Imbalance

NSG223.05.01 Respiratory Acidosis ▪ Treatment aimed at improving ventilation Respiratory Alkalosis ▪ Almost always due to hyperventilation ▪ Manifestations: lightheadedness, inability to concentrate, numbness and tingling, sometimes loss of consciousness ▪ Correct cause of hyperventilation, rebreather mask, breathe into bag Metabolic Acidosis ▪ Causes: Acute: Diabetes, diarrhea ▪ Causes: Chronic – Low Ca levels, Kidney disease ▪ Manifestations: headache, confusion, drowsiness, increased respiratory rate and depth, hyperkalemia, nausea and vomiting, decreased blood pressure, decreased cardiac output, dysrhythmias, shock; if decrease is slow, patient may be asymptomatic until bicarbonate is 15 mEq/L or less ▪ Correct underlying problem, correct imbalance ▪ Serum calcium levels may be low with chronic metabolic acidosis ▪ Must be corrected before treating acidosis ▪ So Bicarbonate may be administered – watch for increased Na levels!) ▪ With acidosis, hyperkalemia may occur as potassium shifts out of cell ▪ As acidosis is corrected, potassium shifts back into cell, potassium levels decrease - Monitor potassium levels

imbalances. Management of Acid-base balance

  • Clinical manifestations of the 4 imbalances

NSG223.01.05 See above

Management of Acid-base balance - ABG interpretation

NSG223.05.01 See above

Management of Acid-base balance - Electrolyte imbalances with ABG imbalances

NSG223.05.01 See above

Hepatitis (A,B,C,D,E) - Modes of transmission

NSG223.06.01 Hepatitis A viral infection ▪ Affects mainly the adult population ▪ But, children in day care are at risk ▪ Transmission: oral-fecal route, unsanitary food, poor hygiene, hand to mouth, sexual activity

Hepatitis (A,B,C,D,E) - prevention

NSG223.06.01 vaccination

Hepatitis (A,B,C,D,E) - Risk Factors

NSG223.06.01 Hepatitis B Viral infection ▪ Transmission: Blood ▪ Who is at risk: IV drug users, multiple partners, getting tattoos, teenagers, male homosexual activity, hemodialysis, healthcare workers, close contact with a carrier ▪ Long incubation period – most people recover spontaneously ▪ Clinical manifestations: insidious, flu-like, respiratory, loss of appetite, aches, malaise, liver tenderness.. can last 1 – 6 months.. ▪ Prevention: prevent transmission, vaccination

Hepatitis - Nutrition

NSG223.06.01 Hepatitis B Viral infection ▪ Prevention: prevent transmission, vaccination ▪ Management: Alpha-interferon (inhibits viral replication), entecavir, tenofovir (antiviral agents), antiemetics, monitor liver enzymes, rest, nutritious meals (include

protein and low fat))

Hepatitis - Medication induced

NSG223.06.01 ▪ The most common cause of hepatitis – 50% of cases in the USA ▪ Cause: multiple medications can cause liver damage – Acetaminophen, rheumatic agents, analgesics, antidepressants, anticonvulsants, etc... ▪ Clinical manifestations: fever, rash, nausea, chills, pruritus, arthralgia, anorexia, nausea, jaundice, dark urine, enlarged and tender liver ▪ Management: stop the medication, high-dose corticosteroids, liver transplant

Pancreatitis - pathophysiology

NSG223.06.02 ▪ Acute: pancreatic duct becomes obstructed, and enzymes back up, causing autodigestion and inflammation of the pancreas ▪ Chronic: progressive inflammatory disorder with destruction of the pancreas; cells are replaced by fibrous tissue; pressure within the pancreas increases, obstructing the pancreatic and common bile ducts ▪ Mild pancreatitis aka interstitial edematous pancreatitis ▪ Severe pancreatitis aka necrotizing pancreatitis

Pancreatitis - clinical manifestations

NSG223.06.02 ▪ Laboratory tests ▪ Serum amylase level high ▪ Serum lipase level high ▪ Liver enzyme levels high ▪ Triglyceride levels ▪ Glucose level ▪ Bilirubin level ▪ Serum calcium level low ▪ ACUTE: ▪ Severe abdominal pain ▪ mid-epigastric ▪ Acute onset – 24-48 hours after eating or alcohol intake ▪ Abdominal and back tenderness ▪ Abdominal distension ▪ Nausea and vomiting*** ▪ Rigid board-like abdomen – pt in danger of hemorrhaging and peritonitis ▪ Low grade fever ▪ Jaundice ▪ Hypotension

Chronic Pancreatitis - Treatment

NSG223.06.03 ▪ Relief of pain and discomfort ▪ Oral care ▪ Care of NGT ▪ Intake and output ▪ VS ▪ Maintain patient safety – often confused ▪ Respiratory – semi-fowler position ▪ Respiratory assessment ▪ Maintain nutrition – dly weight, enteral/parenteral, high protein - low fat diet ▪ Monitor and prevent complications-peritonitis

Cirrhosis - pathophysiology

NSG223.06.04 • Irreversible, progressive deterioration of the liver - Caused by chronic liver disease - Acute viral hepatitis – necrotic cirrhosis - Chronic hepatitis - Long-term obstruction to biliary flow (gallbladder) – biliary cirrhosis - Alcoholism – alcoholic cirrhosis

Cirrhosis - Treatments

NSG223.06.04 Avoid alcohol, important to rest, diet low Na, restricted fluids, bleeding precautions (soft toothbrush) Cirrhosis

  • Clinical manifestations

NSG223.06.04 • May be asymptomatic until severe liver impact - Gradual onset – body compensates... - Early symptoms nonspecific: - Abdominal pain, ankle edema, firm enlarged liver, splenomegaly, intermittent low-grade fever, fatigue, weakness, anorexia, weight loss, spider veins - Decompensated cirrhosis:

  • Ascites, continuous mild fever, epistaxis (nose bleeds), hypotension, jaundice, purpura, esophageal varices, hemorrhoids, deficiency of fat-soluble vitamins, especially vitamin K, mental deterioration (encephalopathy)

Cirrhosis - Treatment

NSG223.06.04 • Management based on the severity off the illness to relieve symptoms – permanent damage, cannot heal the liver!

  • Antacids, H2 Blockers
  • Vitamins and nutritional supplements
  • Avoid alcohol, sodium restriction, fluid restriction
  • Diuretics (K+ sparing e. Spironolactone)
  • Albumin (ascites)
  • Mentation changes –ammonia high - Lactulose
  • Meds with antifibrotic qualities: colchicine, ACE inhibitors, immunosuppressants,...
  • End stage – Milk Thistle (herb)
  • Paracentesis
  • Transjugular intrahepatic portosystemic shunt (TIPS)

▪ Prolonged renal ischemia – anemia, transfusion reaction, trauma, burns ▪ Nephrotoxic agents – certain antibiotics, heavy metals, radiopaque contrast mediums ▪ Infectious processes – acute pyelonephritis, glomerulonephritis ▪ Post-renal: ▪ Urinary tract obstruction, BPH, calculi, tumors, strictures

Acute renal failure - Precautions

NSG223.07.01 Medical management ▪ Identify underlying cause key to treatment ▪ Maintain fluid balance – dly weight, I&O, VS, cardiac monitoring, measurements of central pressures, se and urine concentrations, ▪ Maintain electrolyte balances ▪ K+ - if high treat with sodium polystyrene sulphonate (Kayexalate) orally or enema*** ▪ Correct acid-base imbalance ▪ If medications and antibiotics needed – may have to adjust dose ▪ Evaluate need for dialysis ▪ Diet – high carb, low protein, low K, low phos (no bananas, potatoes, citrus fruits, juices, coffee, or soda)** ▪ Fluid restriction

Chronic renal failure - Clinical manifestations

NSG223.07.02 ▪ Neurologic: ▪ Asterixis (uncontrolled hand flap) ▪ Confusion, disorientation ▪ Restless legs, tremors ▪ Seizures ▪ Weakness, fatigue ▪ Skin ▪ Coarse thinning hair ▪ Dry flaking skin ▪ Gray-bronze kin color ▪ Cardiovascular ▪ Pitting edema ▪ Periorbital edema ▪ Engorged neck veins

▪ Hypertension ▪ Increased K+ ▪ Pulmonary ▪ Crackles ▪ Tachypnea, SOB ▪ Thick sputum ▪ GI ▪ Ammonia odor to breath ▪ Anorexia, nausea, vomiting ▪ GI bleed ▪ Hematologic ▪ Anemia ▪ Thrombocytopenia ▪ Musculoskeletal ▪ Bone pain, bone fractures ▪ Muscle cramp

Chronic renal failure - diagnostics

NSG223.07.02 ▪ History and Physical ▪ Glomerular Filtration Rate - As GFR decreases – BUN and Se Cr increases ▪ Na+ and water retention – edema, heart failure, hypertension ▪ Metabolic acidosis – kidneys unable to excrete acids ▪ Anemia – inadequate erythropoietin production ▪ Calcium low and Phosphate high imbalance – as one increases the other decreases, in ESKD kidney is unable to balance ▪ Poor urine output

Chronic renal failure - Nutrition

NSG223.07.02 ▪ Diet ▪ Protein restricted ▪ Fluid restricted (500-600ml/day + prev day uo) ▪ K+ restricted ▪ Na+ restricted ▪ High Carbohydrate

Chronic renal failure - Treatments

NSG223.07.02 ▪ Pharmacologic: ▪ Calcium and Phosphorus binders ▪ Ca Carbonate (Os-cal) tums – binds with Phos

(these can be manipulated according to patient needs) ▪ Excess fluid is removed via osmosis/ultrafiltration ▪ Heparin is used to prevent clotting ▪ Cleaned blood is returned to the body Peritoneal dialysis ▪ For patients unwilling or unable to have hemodialysis ▪ In PD the membrane that covers the abdominal organs and lines the abdominal wall is used as the “dialyzer” ▪ The patient has a peritoneal catheter inserted (stays in permanently) ▪ Dialysate is connected to the catheter (warm with heat pad to body temp)** not microwave ▪ Dialysate (2-3L) is infused (5-10min) – has time to dwell (depends on pt, a few hours to overnight) – drained (10-20min) ▪ Nursing: ▪ Assess abdomen ▪ Assess s/s of infection (peritonitis) ▪ Use strict aseptic technique ▪ Watch for leaks ▪ Assess the drainage (no blood – should be straw colored) ▪ Watch electrolytes

Dialysis - Safety

NSG223.07.03 ▪ Vascular access – double lumen large catheter – in subclavian, jugular or femoral vein - usually short term option ▪ Nursing – only used for dialysis, aseptic technique, watch for infection, protect ▪ Arteriovenous fistula – joining an artery and a vein, takes time to mature and heal ▪ Arteriovenous graft – created by placing a synthetic graft material between the artery and vein ▪ Nursing – assess for patency (bruit/thrill), do not use arm for BP, IV, blood samples, check dressings not too tight, no restraints, jewelry***

Pharmacological treatment - Complications

NSG223.07.

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Med surg 2 blueprint exam 2

Course: Medical-Surgical Nursing II (NSG 223)

249 Documents
Students shared 249 documents in this course

University: Herzing University

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NSG223 Medical-Surgical II
EXAM 2 STUDY GUIDE
Topic Location Student Notes
Pulmonary embolism
- pathophysiology NSG 223.04.01.01
PE: refers to obstruction of the pulmonary artery or one of its branches by a thrombi
that originates somewhere in the right side of the heart
Causes:
Trauma
Surgery (orthopedic, abdominal, pelvic,..)
Pregnancy
Heart Failure
Hypercoagulable states
Prolonged immobility
Most commonly PE is due to blood clot….but can also be due to air, fat, amniotic
fluid, sepsis (bacterial vegetation)
Complete or partial occlusion
Little or no blood-flow to distal area
Ventilation/Perfusion impairment
Impaired or no gas exchange
Hemodynamic changes – edema, regional vasoconstriction – increased pressures –
can lead to right sided heart failure
Pulmonary embolism
- Clinical manifestations
NSG223.04.01.01 Depends on the size of the thrombus…
Dyspnea
Tachycardia
Bloody sputum
Cough
Chest pain (pleuritic)
Anxiety
Fever
Diaphoresis
Syncope
Pulmonary embolism
- Treatments
NSG223.04.01.02 Emergency:
Nasal O2 immediately to relieve hypoxemia, respiratory distress, central cyanosis…

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