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Spinal Stenosis CASE Pathophysiology - Color

Case diagram of spinal stenosis with cap mr
Course

BS Nursing (BSN)

462 Documents
Students shared 462 documents in this course
Academic year: 2024/2025
Listed booksBiochemistryEssentials of Anatomy and PhysiologySeeley's Anatomy and PhysiologySeeley's Essentials of Anatomy and PhysiologyLos bienes
Uploaded by:
Student
Gail Angeli Romuar
Pontifical and Royal University of Santo Tomas, The Catholic University of the Philippines
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Pathophysiology

NND ROOM 3092

KNOWN CASE OF SPINAL STENOSIS

THORACIC AREA; CAP-MR

Modifiable Risk Factors

AGE: 55 YEARS OLD

Non-Modifiable Risk

Factors

Diagnostic Results

Clinical Manifestations

Diagnosis

Mechanism

Legend:

APPERANCE OF

MALIGNANT CELLS

WITHIN THE BONES

Treatment/ DX Test

FOREIGN TO

IMMUNE CELLS

RELEASE OF

INFLAMMATORY

RESPONSE

INCREASE OF

GROWTH

FACTOCTONS

AROUND THE

BONE

SOFT TISSUE

MASS

ACCUMULATION

AROUND THE

BONE

MALIGNANT

SPINE TUMOR

PET SCAN

Cancer Cell

Remnants

SPINE SURGERY

IMMUNE

SUPPRESION

CHEMOTHERAPY

DEGENERATIVE

BODY CHANGES

NARROWING OF

VERTEBRAL

CANAL

ANATOMICAL

CHANGES POST

OP

SPINAL

COMPRESSION

SPINAL

STENOSIS ON

THORACIC

LEVEL

PERMANENT

NERVE DAMAGE

PARALYSIS ON

LEVEL BELOW

THORACIC

AREA

UPPER BODY

WEAKNESS

HIGH EPIDURAL

PRESSURE

NEUROGENIC

CLAUDICATION

SPEAKING IN

PHRASES

SMOKING

DNA CELL

DAMAGE FROM

TOBACCO

SUPPRESED

NEUTROPHIL

FUNCTION AND

DAMAGED LUNG

EPITHELIUM

SUSCEPTIBLE

HOST AND/ OR

VIRULENT

PATHOGEN

PROLIFERATION

OF MICROBE IN

LOWER

AIRWAYS AND

ALVEOLI

PARALYSIS/ WEAKNESS

OF RESPIRATORY

MUSCLES

SYSTEMATIC

INFLAMMATORY

RESPONSE TOWARDS

INVADING MICROBES

SYSTEMIC CYTOKINE

RELEASE CAUSING

DYSRUPTION IN

HYPOTHALAMIC

THERMOREGULATION

FEVER

PARACETAMOL

INVASIVE CELL

DAMAGE

LOCAL

RESPONSE BY

ALVEOLAR

EPITHELIAL

CELLS

ACCUMULATION OF

NEUTROPHILS AND

PLASMA EXUDATE

IRRITATION AND

ATTEMPTED

CLEARANCE OF

AIRWAYS

FLUID

INFLITRATES

INSIDE ALVEOLI,

PHLEGM

PRODUCTION

PRODUCTIVE

COUGH

FLUID BUILD UP

NOT ALLOWING

X-RAYS TO PASS

THROUGH

CHEST X RAY

CONSOLIDATION

ON CHEST X

RAY

ALVEOLAR SACS

BLOCKED BY

FLUID

ACCUMULATION

DECREASE

EXCHANGE OF

CO2 AND O

TRIGGERS PERIPHERAL AND

CENTRALS CHEMORECEPTORS

TO INCREASE RESPIRATORY

DRIVE

SHORTNESS OF

BREATH

DEEP

BREATHING

EXERCISE

THICKENING OF

ALVEOLAR

WALLS

IRRITATED

ALVEOLAR

WALLS

FLUID INFILTRATES NOT IN

ALVEOLAR, COUGHING DOES NOT

LEAD TO FLUID PRODUCTION

DRY COUGH

CHEST

PHYSIOTHERAPHY

ANTIBIOTIC

THERAPHY

COMPLETELY

ASSISTED FOR

ADLS

UNABLE TO EAT

AND ENJOY

FOOD

METABOLIC

CHANGES

NUTRIOTION

LESS THAN

BODY

REQUIREMENT

BLADDER

/ RENAL

AFFECTATION

LOW

POTASSIUM

INTAKE

LOW SERUM

POTASSIUM: 2.

SERUM K,

SERUM NA TEST

BODY

WEAKNESS

UNSTABLE

BLOOD

PRESSURE

DROWSINES

20 MEQS

POTASSIUM VIA

IV; 2 TABS KCL

TAB

PARENTS

HISTORY OF

HEART ATTACK

DIAGNOSED

WITH

HYPERTENSION

ARRHYTMIAS

Was this document helpful?

Spinal Stenosis CASE Pathophysiology - Color

Course: BS Nursing (BSN)

462 Documents
Students shared 462 documents in this course

University: Pontifical and Royal University of Santo Tomas, The Catholic University of the Philippines

Was this document helpful?
Pathophysiology
NND ROOM 3092
KNOWN CASE OF SPINAL STENOSIS
THORACIC AREA; CAP-MR
Modifiable Risk Factors
AGE: 55 YEARS OLD
Non-Modifiable Risk
Factors
Diagnostic Results
Clinical Manifestations
Diagnosis
Mechanism
Legend:
APPERANCE OF
MALIGNANT CELLS
WITHIN THE BONES
Treatment/ DX Test
FOREIGN TO
IMMUNE CELLS
RELEASE OF
INFLAMMATORY
RESPONSE
INCREASE OF
GROWTH
FACTOCTONS
AROUND THE
BONE
SOFT TISSUE
MASS
ACCUMULATION
AROUND THE
BONE
MALIGNANT
SPINE TUMOR
PET SCAN
Cancer Cell
Remnants
SPINE SURGERY
IMMUNE
SUPPRESION
CHEMOTHERAPY
DEGENERATIVE
BODY CHANGES
NARROWING OF
VERTEBRAL
CANAL
ANATOMICAL
CHANGES POST
OP
SPINAL
COMPRESSION
SPINAL
STENOSIS ON
THORACIC
LEVEL
PERMANENT
NERVE DAMAGE
PARALYSIS ON
LEVEL BELOW
THORACIC
AREA
UPPER BODY
WEAKNESS
HIGH EPIDURAL
PRESSURE
NEUROGENIC
CLAUDICATION
SPEAKING IN
PHRASES
SMOKING
DNA CELL
DAMAGE FROM
TOBACCO
SUPPRESED
NEUTROPHIL
FUNCTION AND
DAMAGED LUNG
EPITHELIUM
SUSCEPTIBLE
HOST AND/ OR
VIRULENT
PATHOGEN
PROLIFERATION
OF MICROBE IN
LOWER
AIRWAYS AND
ALVEOLI
PARALYSIS/ WEAKNESS
OF RESPIRATORY
MUSCLES
SYSTEMATIC
INFLAMMATORY
RESPONSE TOWARDS
INVADING MICROBES
SYSTEMIC CYTOKINE
RELEASE CAUSING
DYSRUPTION IN
HYPOTHALAMIC
THERMOREGULATION
FEVER
PARACETAMOL
INVASIVE CELL
DAMAGE
LOCAL
RESPONSE BY
ALVEOLAR
EPITHELIAL
CELLS
ACCUMULATION OF
NEUTROPHILS AND
PLASMA EXUDATE
IRRITATION AND
ATTEMPTED
CLEARANCE OF
AIRWAYS
FLUID
INFLITRATES
INSIDE ALVEOLI,
PHLEGM
PRODUCTION
PRODUCTIVE
COUGH
FLUID BUILD UP
NOT ALLOWING
X-RAYS TO PASS
THROUGH
CHEST X RAY
CONSOLIDATION
ON CHEST X
RAY
ALVEOLAR SACS
BLOCKED BY
FLUID
ACCUMULATION
DECREASE
EXCHANGE OF
CO2 AND O2
TRIGGERS PERIPHERAL AND
CENTRALS CHEMORECEPTORS
TO INCREASE RESPIRATORY
DRIVE
SHORTNESS OF
BREATH
DEEP
BREATHING
EXERCISE
THICKENING OF
ALVEOLAR
WALLS
IRRITATED
ALVEOLAR
WALLS
FLUID INFILTRATES NOT IN
ALVEOLAR, COUGHING DOES NOT
LEAD TO FLUID PRODUCTION
DRY COUGH
CHEST
PHYSIOTHERAPHY
ANTIBIOTIC
THERAPHY
COMPLETELY
ASSISTED FOR
ADLS
UNABLE TO EAT
AND ENJOY
FOOD
METABOLIC
CHANGES
NUTRIOTION
LESS THAN
BODY
REQUIREMENT
BLADDER
/ RENAL
AFFECTATION
LOW
POTASSIUM
INTAKE
LOW SERUM
POTASSIUM: 2.2
SERUM K,
SERUM NA TEST
BODY
WEAKNESS
UNSTABLE
BLOOD
PRESSURE
DROWSINES
20 MEQS
POTASSIUM VIA
IV; 2 TABS KCL
TAB
PARENTS
HISTORY OF
HEART ATTACK
DIAGNOSED
WITH
HYPERTENSION
ARRHYTMIAS

Students also viewed

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